Mateus Amorim Aboboreira et al. Julio Paiva Maia Vitória Burin Borges Felipe Hideaki Ueda Marcos Franchetti
Introduction
Coronary vasospasm was initially described by Prinzmetal et al. as angina that occurs at rest, without being provoked by an increase in oxygen demand, unlike classic Heberden’s angina, which is induced by physical or emotional stress. The pathophysiology of coronary vasospasm is multifactorial, and various precipitating factors exist.² The present case demonstrates an intriguing scenario that highlights the complexities of clinical management and etiological determination of vasospasm in a post-acute myocardial infarction patient.
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