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  <front>
    <journal-meta>
      <journal-id journal-id-type="publisher-id">dic</journal-id>
      <journal-title-group>
        <journal-title>Arquivos Brasileiros de Cardiologia Imagem cardiovascular</journal-title>
        
        <abbrev-journal-title abbrev-type="publisher">Arq Bras Cardiol: Imagem cardiovasc</abbrev-journal-title>
      </journal-title-group>
      <issn pub-type="epub">2675-312X</issn>
      <publisher>
        <publisher-name>Sociedade Brasileira de Cardiologia</publisher-name>
        
      </publisher>
    </journal-meta>
    <article-meta>
      <article-id pub-id-type="publisher-id">2318-8219.20180026</article-id>
      <article-id pub-id-type="doi">10.5935/2318-8219.20180026</article-id>
      <article-categories>
        <subj-group subj-group-type="heading">
          <subject>Original Article</subject>
        </subj-group>
      </article-categories>
      <title-group>
        <article-title>Correlation between Degrees of Pulmonary Hypertension and Findings in Complementary Tests</article-title>
        <trans-title-group xml:lang="pt">
          <trans-title>Correlação entre Graus de Hipertensão Pulmonar e Achados em Exames Complementares</trans-title>
        </trans-title-group>
      </title-group>
      <contrib-group><contrib contrib-type="author"><name><surname>Pazello</surname><given-names>João Arthur</given-names></name><xref ref-type="aff" rid="aff1"/></contrib><contrib contrib-type="author"><name><surname>Camarozano</surname><given-names>Ana Cristina</given-names></name><xref ref-type="aff" rid="aff1"/></contrib></contrib-group>
      <aff id="aff1"><institution content-type="normalized"/><institution content-type="orgdiv1">Hospital de Clínicas</institution><institution content-type="orgname">Universidade Federal do Paraná</institution><addr-line><named-content content-type="city">Curitiba</named-content><named-content content-type="state">PR</named-content></addr-line><country country="BR">Brazil</country><institution content-type="original">Hospital de Clínicas, Universidade Federal do Paraná, Curitiba, PR - Brazil</institution></aff>
      <author-notes> <corresp id="c01"> Mailing Address: João Arthur Pazello • Rua Ozório Duque Estrada, 226, Jardim Social. Postal Code 82520-470, Curitiba, PR – Brazil E-mail: <email>joao.arthur.pazello@gmail.com</email> </corresp> <fn fn-type="con"> <p>Authors’ contributions</p> <p>Research creation and design: Camarozano AC; Data acquisition: Pazello JA; Data analysis and interpretation: Camarozano AC; Statistical analysis: Pazello JA; Manuscript drafting: Pazello JA; Critical revision of the manuscript as for important intellectual content: Camarozano AC.</p></fn><fn fn-type="conflict">
 <p>Potential Conflicts of Interest</p> <p>There are no relevant conflicts of interest.</p></fn> </author-notes>
      <pub-date date-type="pub" publication-format="electronic">
				<day>20</day>
				<month>01</month>
				<year>2022</year>
			</pub-date>
			<pub-date date-type="collection" publication-format="electronic"> <season>Jul-Sep</season> <year>2018</year> </pub-date>
      <volume>31</volume>
      <issue>3</issue>
      <fpage>175</fpage>
      <lpage>182</lpage>
      <history><date date-type="received"><day>14</day><month>12</month><year>2017</year></date><date date-type="rev-recd"><day>4</day><month>01</month><year>2018</year></date><date date-type="accepted"><day>1</day><month>06</month><year>2018</year></date></history>
      <permissions>
<license license-type="open-access" xlink:href="http://creativecommons.org/licenses/by-nc/3.0/">
<license-p>
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License, which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
</license-p>
</license>
</permissions>
      <abstract><title>Abstract</title><sec><title>Introduction</title><p> Pulmonary hypertension is potentially fatal and courses with important day-to-day limitation. While the treatment is capable of slowing the disease’s progression, increase in life expectancy is directly linked to early diagnosis and treatment.</p></sec><sec><title>Objectives</title><p> To analyze the relation between alterations detectable on routine exams performed on pulmonary hypertension patients (echocardiography, electrocardiography and computerized tomography) and increases on pulmonary artery systolic pressure.</p></sec><sec><title>Methods</title><p> We analyzed the recordings of patients presented with pulmonary hypertension, and separated them in two groups based on their echocardiography-estimated pulmonary artery systolic pressure. Group 1 was composed of patients with pulmonary artery systolic pressure between 35 mmHg and 65 mmHg, and Group 2 of patients with pulmonary artery systolic pressure &gt; 65 mmHg. We analyzed the prevalence of alterations suggestive of pulmonary hypertension on echocardiography, electrocardiography and computerized tomography. A descriptive analysis of the findings was conducted, followed by comparative analysis between the groups.</p></sec><sec><title>Results</title><p> We analyzed 101 patients (43 from Group 1 and 58 from Group 2). Most were women (82). Higher pulmonary artery systolic pressure values were correlated with right-heart dilation on echocardiography (p &lt; 0.001). Electrocardiography findings revealed that right-heart hypertrophy and ventricular strain pattern were more common on Group 2 (p &lt; 0.05). Computerized tomography analysis showed Group 2 patients had superior pulmonary artery diameters (p &lt; 0.05). There was correlation between high pulmonary artery systolic pressure and clinical progression of the disease measured via the NYHA score (p &lt; 0.05).</p></sec><sec><title>Conclusion</title><p> It is possible to make use of clinical examination and a simple electrocardiography to stratify the severity of a pulmonary artery patient. Computerized tomography and echocardiography should be used to confirm these findings. (Arq Bras Cardiol: Imagem cardiovasc. 2018;31(3):175-182)</p></sec></abstract>
      <trans-abstract xml:lang="pt"><title>Resumo</title><sec><title>Fundamento</title><p> A hipertensão pulmonar é potencialmente fatal, com grande impacto na qualidade de vida do paciente.O tratamento pode alterar sua progressão, mas o aumento na sobrevida é relacionado ao diagnóstico e à terapêutica precoces.</p></sec><sec><title>Objetivo</title><p> Analisar a relação entre alterações em exames usados de rotina (ecocardiografia, eletrocardiografia e tomografia computadorizada) e o aumento da pressão sistólica da artéria pulmonar.</p></sec><sec><title>Métodos</title><p> Avaliamos fichas de pacientes portadores de hipertensão pulmonar, separando-os em dois grupos de acordo com a pressão sistólica da artéria pulmonar estimada à ecocardiografia. O Grupo 1 consistiu de pacientes com pressão sistólica da artéria pulmonar entre 35 e 65 mmHg, e o Grupo 2, de pacientes com pressão sistólica da artéria pulmonar &gt; 65 mmHg. Analisamos a prevalência de alterações sugestivas de hipertensão pulmonar em exames de ecocardiografia, eletrocardiografia e tomografia computadorizada. Foram realizadas análises descritiva dos dados e comparativa entre os grupos.</p></sec><sec><title>Resultados</title><p> Foram analisados 101 pacientes (43 do Grupo 1 e 58 do Grupo 2). A maioria foram mulheres (82). Valores maiores de pressão sistólica da artéria pulmonar foram correlacionados com dilatação de câmaras direitas à ecocardiografia (p &lt; 0,001). À eletrocardiografia, sinais de hipertrofia direita e presença de padrão strain ventricular foram mais prevalentes no Grupo 2 (p &lt; 0,05). À tomografia computadorizada, observaram-se maiores diâmetros de tronco da artéria pulmonar em pacientes do Grupo 2 (p &lt; 0,05). Houve correlação entre progressão clínica da doença pelo escore e aumento da pressão sistólica da artéria pulmonar (p &lt; 0,05).</p></sec><sec><title>Conclusão</title><p> É possível, em um primeiro momento, fazer uso do exame clínico e da eletrocardiografia para uma estimativa da gravidade do quadro de hipertensão pulmonar. Os achados, então, devem ser confirmados com análise secundária por tomografia computadorizada e ecocardiografia. (Arq Bras Cardiol: Imagem cardiovasc. 2018;31(3):175-182)</p></sec></trans-abstract>
      <kwd-group xml:lang="en"><kwd>Hypertension, Pulmonary</kwd><kwd>Echocardiography/methods</kwd><kwd>Electrocardiography</kwd><kwd>Tomography, X-Ray Computed</kwd><kwd>Diagnostic Imaging</kwd><kwd>Blood Pressure</kwd><kwd>Pulmonary Arterial</kwd></kwd-group>
      <kwd-group xml:lang="pt"><kwd>Hipertensão Pulmonar</kwd><kwd>Ecocardiografia/métodos</kwd><kwd>Eletrocardiografia</kwd><kwd>Tomografia Computadorizada por Raios X</kwd><kwd>Diagnóstico por Imagem</kwd><kwd>Pressão Sistólica</kwd><kwd>Artéria Pulmonar</kwd></kwd-group>
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